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Items: 3

1.

CFTR Represses a PDX1 Axis to Govern Pancreatic Ductal Cell Fate [Bulk ATAC-seq]

(Submitter supplied) Inflammation, acinar cell destruction, and ductal cell hyperplasia drive pancreatic remodeling in newborns with cystic fibrosis (CF) lacking a functional CFTR channel. In neonatal CF ferrets, these changes are associated with a transient phase of glucose intolerance that involves islet destruction and subsequent regeneration near hyperplastic ducts. Little is known about the phenotypic changes in CF ductal epithelium and its potential impact on islet function. more...
Organism:
Mustela putorius furo
Type:
Genome binding/occupancy profiling by high throughput sequencing
Platform:
GPL34388
7 Samples
Download data: BED
Series
Accession:
GSE264021
ID:
200264021

2.

Illumina HiSeq 4000 (Mustela putorius furo)

Organism:
Mustela putorius furo
2 Series
13 Samples
Download data
Platform
Accession:
GPL34388
ID:
100034388

3.

ATAC WT, donor2

Organism:
Mustela putorius furo
Source name:
Polarized pancreatic ductal epithelia
Platform:
GPL34388
Series:
GSE264021
Download data: BED
Sample
Accession:
GSM8208878
ID:
308208878

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db=gds|term=GSM8208878[Accession]|query=1|qty=2|blobid=MCID_674e16de8c9ed417fb09038e|ismultiple=true|min_list=5|max_list=20|def_tree=20|def_list=|def_view=|url=/Taxonomy/backend/subset.cgi?|trace_url=/stat?
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