Similar studies for GEO DataSets (Select 2216) - GEO DataSets

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Items: 3

Select item 22161.

Lipopolysaccharide antagonist effect on lipopolysaccharide-stimulated dendritic cells: time course

Analysis of monocyte-derived dendritic cells treated with cyanobacterial product (CyP) for 1 or 3 hours, or with lipopolysaccharide (LPS) in the presence or absence of CyP for 3 hours. Results provide insight into the extent of the antagonistic activity of CyP on LPS-induced responses.

Organism:: Homo sapiens

Type:: Expression profiling by array, count, 4 agent, 2 time sets

Platform:: GPL570
Series:: GSE4748
10 Samples

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DataSet

Accession:: GDS2216

ID:: 2216

PubMed Full text in PMC Similar studies GEO Profiles Analyze DataSet

Select item 2000047482.

A cyanobacterial LPS antagonist prevents endotoxin shock and blocks sustained TLR4 stimulation

(Submitter supplied) A cyanobacterial LPS antagonist prevents endotoxin shock and blocks sustained TLR4 stimulation required for cytokine expression. We report the identification and biologic characterization of an LPS-like molecule extracted from the cyanobacterium Oscillatoria Planktothrix FP1 (CyP). Keywords: different ligands

Organism:: Homo sapiens

Type:: Expression profiling by array

Dataset:: GDS2216
Platform:: GPL570
10 Samples

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Series

Accession:: GSE4748

ID:: 200004748

PubMed Full text in PMC Similar studies Analyze with GEO2R

Select item 2001013963.

Next generation sequencing of human monocyte derived dendritic cells stimulated for two hours with LPS, C5a and/or MSK inhibitor SB-747561

(Submitter supplied) In order to determine the full breath of C5aR and TLR crosstalk in human DC, RNA sequencing analysis on human moDC stimulated in the absence or presence of C5a and TLR4 ligand lipopolysaccharide (LPS) was performed. To minimize interference of induced positive and negative feedback loops, we analyzed gene expression after two hours of DC activation. The involvement of CREB signaling in C5aR and TLR crosstalk was investigated by including moDC stimulated in the presence of MSK inhibitor.