| Circular RNA circEfnb2 promotes cell injury after cerebral infarction by sponging miR-202-5p and regulating TRAF3 expression. | Circular RNA circEfnb2 promotes cell injury after cerebral infarction by sponging miR-202-5p and regulating TRAF3 expression.
Tu L, Cheng W, Wang X, Li Z, Li X. | 09/20/2024 |
| MiR-202-3p Targets Calm1 and Suppresses Inflammation in a Mouse Model of Acute Respiratory Distress Syndrome. | MiR-202-3p Targets Calm1 and Suppresses Inflammation in a Mouse Model of Acute Respiratory Distress Syndrome.
Liu Y, Li H, Pang Y, Li Y, Li S. | 08/26/2024 |
| Bone marrow mesenchymal stem cell-derived exosomes miR-202-5p inhibited pyroptosis to alleviate lung ischemic-reperfusion injury by targeting CMPK2. | Bone marrow mesenchymal stem cell-derived exosomes miR-202-5p inhibited pyroptosis to alleviate lung ischemic-reperfusion injury by targeting CMPK2.
Sun ZL, You T, Zhang BH, Liu Y, Liu J. | 09/12/2023 |
| MicroRNA-202-5p-dependent inhibition of Bcl-2 contributes to macrophage apoptosis and atherosclerotic plaque formation. | MicroRNA-202-5p-dependent inhibition of Bcl-2 contributes to macrophage apoptosis and atherosclerotic plaque formation.
Xu F, Yao F, Ning Y. | 04/26/2023 |
| MicroRNA-202 safeguards meiotic progression by preventing premature SEPARASE-mediated REC8 cleavage. | MicroRNA-202 safeguards meiotic progression by preventing premature SEPARASE-mediated REC8 cleavage.
Chen J, Gao C, Luo M, Zheng C, Lin X, Ning Y, Ma L, He W, Xie D, Liu K, Hong K, Han C., Free PMC Article | 08/13/2022 |
| The microRNA miR-202 prevents precocious spermatogonial differentiation and meiotic initiation during mouse spermatogenesis. | The microRNA miR-202 prevents precocious spermatogonial differentiation and meiotic initiation during mouse spermatogenesis.
Chen J, Gao C, Lin X, Ning Y, He W, Zheng C, Zhang D, Yan L, Jiang B, Zhao Y, Alim Hossen M, Han C. | 02/12/2022 |
| miR-202-3p overexpression attenuates endometriosis-like lesions by modulating YAP-dependent transcription of S100A6 in murine models. | miR-202-3p overexpression attenuates endometriosis-like lesions by modulating YAP-dependent transcription of S100A6 in murine models.
Lan J, Xie K. | 01/16/2021 |
| A significant decrease in miR-202 expression was observed in Non-Small Cell Lung Cancer cells both in vivo and in vitro. In addition, miR-202 expression was associated with drug resistance. | miR-202 Enhances the Anti-Tumor Effect of Cisplatin on Non-Small Cell Lung Cancer by Targeting the Ras/MAPK Pathway.
Sun W, Ping W, Tian Y, Zou W, Liu J, Zu Y. | 01/26/2019 |
| expression of miR-202 was different in the skin tissue of C57BL/6 black mice and BALB/c white mice and that wnt5a, kit, and tcf7 are negatively regulated by miR-202 | Differential Expression of miR-202 and Validation of Predicted Target Genes in the Skin Tissue of C57BL/6 Black Mice and BALB/c White Mice.
Qu L, Li J, Zhao Z, Jiang H, Zhang Q. | 09/9/2017 |
| The results have revealed a miR-202-centered regulatory network that controls the fate of spermatogonial stem cells. | MicroRNA-202 maintains spermatogonial stem cells by inhibiting cell cycle regulators and RNA binding proteins.
Chen J, Cai T, Zheng C, Lin X, Wang G, Liao S, Wang X, Gan H, Zhang D, Hu X, Wang S, Li Z, Feng Y, Yang F, Han C., Free PMC Article | 08/26/2017 |
| This study provides novel insight into the role of miRNA in cerebellar degeneration and suggests that miR-202 is a key miRNA mediating the pathogenesis of multiple-system atrophy. | Altered expression of miR-202 in cerebellum of multiple-system atrophy.
Lee ST, Chu K, Jung KH, Ban JJ, Im WS, Jo HY, Park JH, Lim JY, Shin JW, Moon J, Lee SK, Kim M, Roh JK. | 10/24/2015 |
| expression of the conserved gonad microRNA, miR-202-5p/3p, is downstream of the testis-determining factor SOX9, suggesting an early role in testis development. | SOX9 regulates microRNA miR-202-5p/3p expression during mouse testis differentiation.
Wainwright EN, Jorgensen JS, Kim Y, Truong V, Bagheri-Fam S, Davidson T, Svingen T, Fernandez-Valverde SL, McClelland KS, Taft RJ, Harley VR, Koopman P, Wilhelm D. | 10/26/2013 |