| MiR-29c alleviates hyperglycemia-induced inflammation via targeting TGF-beta in cardiomyocytes. | MiR-29c alleviates hyperglycemia-induced inflammation via targeting TGF-β in cardiomyocytes.
Zhong H, Tang H, Wang Y, Tang S, Zhu H. | 08/23/2024 |
| Impact of miR-29c-3p in the Nucleus Accumbens on Methamphetamine-Induced Behavioral Sensitization and Neuroplasticity-Related Proteins. | Impact of miR-29c-3p in the Nucleus Accumbens on Methamphetamine-Induced Behavioral Sensitization and Neuroplasticity-Related Proteins.
Su H, Zhu L, Su L, Li M, Wang R, Zhu J, Chen Y, Chen T., Free PMC Article | 06/21/2024 |
| Dissecting the Long-Term Effect of Stress Early in Life on FKBP5: The Role of miR-20b-5p and miR-29c-3p. | Dissecting the Long-Term Effect of Stress Early in Life on FKBP5: The Role of miR-20b-5p and miR-29c-3p.
Cattane N, Di Benedetto MG, D'Aprile I, Riva MA, Cattaneo A., Free PMC Article | 04/2/2024 |
| miR-29c-3p Accelerates Mucosal Repair in Dextran Sodium Sulfateinduced Ulcerative Colitis Mice through the KDM6B/H3K27me3/LDHA Axis. | miR-29c-3p Accelerates Mucosal Repair in Dextran Sodium Sulfateinduced Ulcerative Colitis Mice through the KDM6B/H3K27me3/LDHA Axis.
Li X, Yin C, Li J. | 07/21/2023 |
| MiR-29c Inhibits TNF-alpha-Induced ROS Production and Apoptosis in Mouse Hippocampal HT22 Cell Line. | MiR-29c Inhibits TNF-α-Induced ROS Production and Apoptosis in Mouse Hippocampal HT22 Cell Line.
Li B, Lu Y, Wang R, Xu T, Lei X, Jin H, Gao X, Xie Y, Liu X, Zeng J. | 02/11/2023 |
| MicroRNA miR-29c regulates RAG1 expression and modulates V(D)J recombination during B cell development. | MicroRNA miR-29c regulates RAG1 expression and modulates V(D)J recombination during B cell development.
Kumari R, Roy U, Desai S, Nilavar NM, Van Nieuwenhuijze A, Paranjape A, Radha G, Bawa P, Srivastava M, Nambiar M, Balaji KN, Liston A, Choudhary B, Raghavan SC. | 02/12/2022 |
| MiR-29c-3p May Promote the Progression of Alzheimer's Disease through BACE1. | MiR-29c-3p May Promote the Progression of Alzheimer's Disease through BACE1.
Cao Y, Tan X, Lu Q, Huang K, Tang X, He Z., Free PMC Article | 01/15/2022 |
| miR-29c-3p regulates TET2 expression and inhibits autophagy process in Parkinson's disease models. | miR-29c-3p regulates TET2 expression and inhibits autophagy process in Parkinson's disease models.
Wang R, Yao J, Gong F, Chen S, He Y, Hu C, Li C. | 01/1/2022 |
| MiR-29c protects against inflammation and apoptosis in Parkinson's disease model in vivo and in vitro by targeting SP1. | MiR-29c protects against inflammation and apoptosis in Parkinson's disease model in vivo and in vitro by targeting SP1.
Wang R, Yang Y, Wang H, He Y, Li C. | 05/8/2021 |
| High-throughput screening identified miR-7-2-3p and miR-29c-3p as metastasis suppressors in gallbladder carcinoma. | High-throughput screening identified miR-7-2-3p and miR-29c-3p as metastasis suppressors in gallbladder carcinoma.
Lu K, Feng F, Yang Y, Liu K, Duan J, Liu H, Yang J, Wu M, Liu C, Chang Y. | 05/1/2021 |
| Circ_016719 plays a critical role in neuron cell apoptosis induced by I/R via targeting miR-29c/Map2k6. | Circ_016719 plays a critical role in neuron cell apoptosis induced by I/R via targeting miR-29c/Map2k6.
Tang C, Ou J, Kou L, Deng J, Luo S. | 01/2/2021 |
| miR-29c-3p inhibits microglial NLRP3 inflammasome activation by targeting NFAT5 in Parkinson's disease. | miR-29c-3p inhibits microglial NLRP3 inflammasome activation by targeting NFAT5 in Parkinson's disease.
Wang R, Li Q, He Y, Yang Y, Ma Q, Li C. | 10/24/2020 |
| he results herein suggest that miR-29c can improve skeletal muscle size and function by stimulating satellite cell proliferation and repressing atrophy-related genes. | miR-29c improves skeletal muscle mass and function throughout myocyte proliferation and differentiation and by repressing atrophy-related genes.
Silva WJ, Graça FA, Cruz A, Silvestre JG, Labeit S, Miyabara EH, Yan CYI, Wang DZ, Moriscot AS., Free PMC Article | 08/12/2020 |
| HIF-1-regulation of miR-29c reduces SERCA2 expression and cardiac contractility. | HIF-1 regulation of miR-29c impairs SERCA2 expression and cardiac contractility.
Williams AL, Walton CB, MacCannell KA, Avelar A, Shohet RV., Free PMC Article | 02/15/2020 |
| results suggest that miR-29c is an important epigenetic regulator of drug-induced behavioural sensitization and changes in gene expression. | Regulation of microRNA-29c in the nucleus accumbens modulates methamphetamine -induced locomotor sensitization in mice.
Su H, Zhu L, Li J, Wang R, Liu D, Han W, Cadet JL, Chen T. | 12/28/2019 |
| Canonical TGF-beta signaling promotes senescence via miR-29-induced loss of H4K20me3. Mechanistically, oxidative stress triggers TGF-beta signaling. Activated TGF-beta signaling gives rise to acute accumulation of miR-29a and miR-29c both of which directly suppress their novel target, Suv4-20h. | TGF-β signaling alters H4K20me3 status via miR-29 and contributes to cellular senescence and cardiac aging.
Lyu G, Guan Y, Zhang C, Zong L, Sun L, Huang X, Huang L, Zhang L, Tian XL, Zhou Z, Tao W., Free PMC Article | 01/19/2019 |
| these findings suggest that miR-29c is a negative regulator of axonal growth of dorsal root ganglia neurons by targeting PRKCI under hyperglycemia. | MiR-29c/PRKCI Regulates Axonal Growth of Dorsal Root Ganglia Neurons Under Hyperglycemia.
Jia L, Wang L, Chopp M, Li C, Zhang Y, Szalad A, Zhang ZG., Free PMC Article | 12/22/2018 |
| The present study demonstrated that TUG1, along with miR29c, may contribute to cardiac FMT activation and promote fibrosis in chronic hypoxia. | Long noncoding RNA TUG1 promotes cardiac fibroblast transformation to myofibroblasts via miR‑29c in chronic hypoxia.
Zhu Y, Feng Z, Jian Z, Xiao Y. | 11/10/2018 |
| Deletion of miR-29 prevents cardiac hypertrophy and fibrosis and improves cardiac function in a mouse model. | Cardiac myocyte miR-29 promotes pathological remodeling of the heart by activating Wnt signaling.
Sassi Y, Avramopoulos P, Ramanujam D, Grüter L, Werfel S, Giosele S, Brunner AD, Esfandyari D, Papadopoulou AS, De Strooper B, Hübner N, Kumarswamy R, Thum T, Yin X, Mayr M, Laggerbauer B, Engelhardt S., Free PMC Article | 09/15/2018 |
| PU.1 suppresses Sirt1 translation via transcriptional promotion of miR-34a/-29c. | PU.1-deficient mice are resistant to thioacetamide-induced hepatic fibrosis: PU.1 finely regulates Sirt1 expression via transcriptional promotion of miR-34a and miR-29c in hepatic stellate cells.
Liu Q, Zhang Y, Yang S, Wu Y, Wang J, Yu W, Liu Y., Free PMC Article | 07/21/2018 |
| miR-29 directly targeted SPARC, resulting in degradation of SPARC-encoding mRNA and reduction in the SPARC protein level. | MiR-29 family members interact with SPARC to regulate glucose metabolism.
Song H, Ding L, Zhang S, Wang W. | 05/12/2018 |
| miR-29c maintains epithelial integrity and promotes recovery from lung injury, thereby attenuating lung fibrosis | MicroRNA-29c Prevents Pulmonary Fibrosis by Regulating Epithelial Cell Renewal and Apoptosis.
Xie T, Liang J, Geng Y, Liu N, Kurkciyan A, Kulur V, Leng D, Deng N, Liu Z, Song J, Chen P, Noble PW, Jiang D., Free PMC Article | 12/9/2017 |
| miR29c stimulation during influenza virus entry enhances the ability of dendritic cells to activate lymphocytes and secrete cytokines IL-6 or TNF-alpha. | H9N2 avian influenza virus enhances the immune responses of BMDCs by down-regulating miR29c.
Lin J, Xia J, Chen YT, Zhang KY, Zeng Y, Yang Q. | 12/9/2017 |
| MiR-29c represses active DNA demethylation enzymes TET and TDG during somatic cell reprogramming | MicroRNA-29 impairs the early phase of reprogramming process by targeting active DNA demethylation enzymes and Wnt signaling.
Fráguas MS, Eggenschwiler R, Hoepfner J, Schiavinato JLDS, Haddad R, Oliveira LHB, Araújo AG, Zago MA, Panepucci RA, Cantz T. | 11/25/2017 |
| miR-29c upregulation or miR-129-5p downregulation effectively prevented epithelial-mesenchymal transition (EMT) in biliary atresia by regulating the expression of EMT pathway-related proteins. | Effect of miR-29c and miR-129-5p on epithelial-mesenchymal transition in experimental biliary atresia mouse models.
Wang JY, Li ZH, Ye M, Feng Q, Chen ZM, Ye XS, Wu ZG, Wang B, Liu L, Yao J. | 02/25/2017 |