Warning: The NCBI web site requires JavaScript to function. more...
An official website of the United States government
The .gov means it's official. Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you're on a federal government site.
The site is secure. The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.
Inhibition of goat lens AR assessed as gibbs binding free energy
Assay data:1 Tested
SummaryPubMed Citation
Inhibition of goat lens AR at 100 uM incubated for 10 mins in presence of NADPH by spectroscopic analysis
Assay data:7 Tested
Inhibition of goat 5-LOX by spectrophotometric analysis
Cytotoxicity against goat fetal fibroblast cells incubated for 48 hrs by MTT assay
Assay data:1 Active, 6 Tested
SummaryCompounds, ActivePubMed Citation
Binding affinity to goat tubulin by double reciprocal plot analysis
Assay data:2 Tested
Inhibition of goat brain tubulin polymerization
Assay data:2 Active, 2 Tested
Binding affinity to goat tubulin assessed as increase in tubulin-colchicine fluorescence preincubated for 30 mins followed by colchicine addition measured after 60 mins by tryptophan fluorescence quenching assay
Binding affinity to goat tubulin by tryptophan fluorescence quenching assay
Assay data:1 Active, 1 Tested
Protection against CuCl2/ascorbic acid-induced depolarization of goat cardiac mitochondria at 66 uM after 60 mins by calcein AM dye-based flow cytometric analysis
Toxicity in goat cardiac mitochondria assessed as effect on calcium accumulation at 66 uM after 60 mins by calcein AM dye-based flow cytometric analysis
Protective activity in goat cardiac mitochondria assessed as reduction in CuCl2/ascorbic acid-induced calcium accumulation at 66 uM after 60 min (Rvb = 25.32%)
Induction of structural distortion in goat cardiac mitochondria at 66 uM after 1 hr by scanning electron microscopy
Protective activity in goat cardiac mitochondria assessed as inhibition of CuCl2/ascorbic acid-induced perforated surface at 66 uM incubated for 1 hr by scanning electron microscopy
Protective activity in goat cardiac mitochondria assessed as inhibition of CuCl2/ascorbic acid-induced convoluted membranes at 66 uM incubated for 1 hr by scanning electron microscopy
Effect on cytochrome c oxidoreductase activity in goat cardiac mitochondria at 8.25 to 66 uM incubated for 1 hr and measured at 10 secs time interval for 90 secs by spectrophotometric analysis
Effect on cytochrome c oxidase activity in goat cardiac mitochondria at 8.25 to 66 uM incubated for 1 hr and measured at 10 secs time interval for 90 secs by spectrophotometric analysis
Protective activity against CuCl2/ascorbic acid-induced apoptotic alteration in goat cardiac mitochondria assessed as reversal of CuCl2/ascorbic acid-induced reduction in cytochrome c oxidase activity at 66 uM measured at 10 secs time interval for 90 secs by spectrophotometric analysis relative to control
Protective activity against CuCl2/ascorbic acid-induced apoptotic alteration in goat cardiac mitochondria assessed as reversal of CuCl2/ascorbic acid-induced reduction in cytochrome c oxidase activity at 33 uM incubated for 1 hr and measured at 10 secs time interval for 90 secs by spectrophotometric analysis relative to control
Protective activity against CuCl2/ascorbic acid-induced apoptotic alteration in goat cardiac mitochondria assessed as reversal of CuCl2/ascorbic acid-induced reduction in cytochrome c oxidase activity at 16.5 uM incubated for 1 hr and measured at 10 secs time interval for 90 secs by spectrophotometric analysis relative to control
Protective activity against CuCl2/ascorbic acid-induced apoptotic alteration in goat cardiac mitochondria assessed as reversal of CuCl2/ascorbic acid-induced reduction in cytochrome c oxidase activity at 8.25 uM and measured at 10 secs time interval for 90 secs by spectrophotometric analysis (Rvb = 72.41%)
Filters: Manage Filters
Your browsing activity is empty.
Activity recording is turned off.
Turn recording back on