optic atrophy 1, isoform C [Drosophila melanogaster]

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ATF4-dependent increase in mitochondrial-endoplasmic reticulum tethering following OPA1 deletion in skeletal muscle. [J Cell Physiol. 2024]
ATF4-dependent increase in mitochondrial-endoplasmic reticulum tethering following OPA1 deletion in skeletal muscle.
Hinton A Jr, Katti P, Mungai M, Hall DD, Koval O, Shao J, Vue Z, Lopez EG, Rostami R, Neikirk K, et al. J Cell Physiol. 2024 Apr; 239(4):e31204. Epub 2024 Feb 28.
Mitochondrial dysfunction in optic neuropathies: animal models and therapeutic options. [Curr Opin Neurol. 2013]
Mitochondrial dysfunction in optic neuropathies: animal models and therapeutic options.
Carelli V, La Morgia C, Sadun AA. Curr Opin Neurol. 2013 Feb; 26(1):52-8.
Heterozygous mutation of Drosophila Opa1 causes the development of multiple organ abnormalities in an age-dependent and organ-specific manner. [PLoS One. 2009]
Heterozygous mutation of Drosophila Opa1 causes the development of multiple organ abnormalities in an age-dependent and organ-specific manner.
Shahrestani P, Leung HT, Le PK, Pak WL, Tse S, Ocorr K, Huang T. PLoS One. 2009 Aug 31; 4(8):e6867. Epub 2009 Aug 31.

RefSeq mRNA
See reference mRNA sequence for the Opa1 gene (NM_001299482.1).
RefSeq protein isoforms
See 5 reference sequence protein isoforms for the Opa1 gene.

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GlyGen glycoinformatics resource [GlyGen glycoinformatics resou...]
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Protein